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Even when ingested in normal amounts, copper and iron may become toxic in certain individuals with genetic predispositions that lead to hepatic overload - iron overload in hemochromatosis [11-17] or copper overload in Wilson disease [18-27]

The compound in solution is sensitive to oxidation, pH, and temperature

Ts1Cje, a partial trisomy 16 mouse model for Down syndrome, exhibits learning and behavioral abnormalities

[8] The same copper that drives the biology is the primary safety liability systemically, and the therapeutic window enough copper to help, not enough to harm is uncharacterized for this route in humans

Therefore, it is crucial to elucidate the composition of trace elements in cardiovascular diseases relative to physiological levels and provide valuable insights into treatment strategies for cardiovascular diseases